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THE PATIENT RELATIONS CONSULTATION PROCESS From: The Texas University Houston Psychiatric Hospital Policies and Procedures Manual Stage 1 The patient contacts Patient Relations by writing or calling or through the hospital staff. Confidentiality: When staff helps a patient contact Patient Relations, the information s he provides is confidential. Stage 2 WHEN.The complaint involves rights issues THEN.Patient Relations does the following: Determines if it is necessary to meet Schedules a meeting within a work day Stage 3 From the patient's complaint, Patient Relations determines and provides the patient with all the necessary information regarding hospital staff members who are the source to answer the concern. Stage 4 Patient Relations determines the need for further actions based on the information collected during its meetings. Examples: Notification of parents, guardians, or family members Stage 5 Does the complaint involve a hospital staff member? If yes, Patient Relations informs the Medical Director or appropriate Director Manager of all the relevant information to determine if the complaint involves clinical practice issues If no, Patient Relations pursues investigation Stage 6 Upon receipt of a decision, do any unresolved issues remain? If yes, Patient Relations forwards to the appropriate Director Manager. If no, Patient Relations delivers appropriate information, in writing, to the patient Stage 7 Patient Relations documents all its services to the patient including the final report, informs patient, and maintains the documentation in a locked file and in the Resolve database.
When corporate or governmental wrongdoing threatens our health and safety, or violates the fundamental principles of fairness and justice, CoPIRG stands up for the public interest in Colorado. We conduct investigative research, publish reports and exposs, advocate new laws, and, when necessary, take corporate wrongdoers or unresponsive government to court. CoPIRG was founded in 1973, and has offices in Denver, Boulder and a national lobbying office in Washington, D.C, for example, jprofiler.
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EXHIBIT B DISCLOSURE AND RELEASE AGREEMENT RELATED TO THE EXCHANGE OF NON-PLAN OPTIONS FOR STOCK OPTIONS UNDER THE NOVADEL PHARMA INC. 1998 STOCK OPTION PLAN You entered into a Non-Plan Nonqualified Stock Option Agreement with NovaDel Pharma Inc. the "Company" ; on February 28, 2005 Agreement" ; . Under the terms of the 2005 Agreement you were issued an option under a "nonqualified non-plan" in FIFTY THOUSAND 50, 000 ; SHARES of the Company's common stock, par value $.001 per share. The Company has since determined that the Company's issuance of such options could give rise to compliance issues regarding the continued listing standards of the American Stock Exchange and the Company therefore desires to cancel the non-plan options issued since May 11, 2004, under the nonqualified non-plan. The Company proposes to issue you new options under the existing NovaDel Pharma Inc. 1998 Stock Option Plan "1998 Plan" ; . The terms of the 1998 Plan and the form agreement thereto are not identical to the terms of the 2005 Agreement. The below summary outlines the differences between the 2005 Agreement and the 1998 Plan Stock Option Agreement that the Company proposes to enter into with you "Agreement" ; . Unless otherwise defined herein, all capitalized terms are defined in either the Agreement or the 1998 Plan. You should carefully read the 2005 Agreement and the 1998 Plan attached to your Agreement and consider all of the information provided in this Disclosure. You are encouraged to consult your own legal counsel before entering into the Agreement offered to you along with this Disclosure and Release. SUMMARY OF YOUR 1998 PLAN STOCK OPTION AGREEMENT 1. 2. 3. You are granted an option for fifty thousand shares of the Company's common stock. The Option is for the same number of shares and is the same class of the Company's stock as your existing option under the 2005 Agreement "Non-Plan Option" ; . The exercise price is $1.41 for each share of common stock you purchase under the Agreement. The exercise price is the same exercise price as under your 2005 Agreement. Your Option under the 1998 Plan will have the same vesting schedule as your Non-Plan Option. Your vesting schedule is: a. b. c. 16, 666 Option shares vest on or after February 27, 2006; 16, Option shares vest on or after February 27, 2007; 16, Option shares vest on or after February 27, 2008 and oxybutynin.
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Global Ophthalmic Market The total market size for prescription ophthalmic drugs in the US alone amounted to $2.8 billion in 2002. An aging population as well as a growing awareness of patients and health care personnel are expected to drive towards increasing ophthalmic pharmaceutical sales. Leading products in this market include Tobradex for steroid-responsive inflammatory ocular conditions, including certain infective conjunctivitis, chronic anterior uveitis and corneal injury and post-operative cataract, with global6 sales of nearly $200 million in 2002. Restasis, indicated for dry eye disease, represented $38 million global6 sales in 2003. Leading drugs in the allergic conjunctivitis area include Patanol $198 million7 in 2002 sales ; , Acular $766 million in 2002 sales ; and Ocufllox $946 million in 2002 sales.
291. Gram, L. & D. Schmidt: Innovative designs of controlled clinical trials in epilepsy. Epilepsia 34 Suppl 7, S1-S6 1993 ; 292. Beydoun, A.: Monotherapy trials of new antiepileptic drugs. Epilepsia 38 Suppl 9, S21-S31, 1997 ; 293. Chadwick, D.: Monotherapy clinical trials of new anti-epileptic drugs: design, indications, and controversies. Epilepsia 38 Suppl 9, S16-S20 1997 ; 294. Perucca, E.: Innovative monotherapy trial designs for the assessment of anti-epileptic drugs: a critical appraisal. Eur J Clin Pharmacol 54, 1-5 1998 ; 295. Devinsky, O., R.E. Faught, B.J. Wilder, A.M. Kanner, M. Kamin, L.D. Kramer & A. Rosenberg: Efficacy of felbamate monotherapy in patients undergoing presurgical evaluation of partial seizures. Epilepsy Res 20, 241-246 1995 ; 296. Bourgeois, B.F., I.E. Leppik, J.C. Sackellares, K. Laxer, R. Lesser, J.A. Messenheimer, L.D. Kramer, M. Kamin & A. Rosenberg: Felbamate: a double-blind controlled trial in patients undergoing presurgical evaluation of partial seizures. Neurology 43, 693-696 1993 ; 297. Theodore, W.H., P. Albert, B. Stertz, B. Malow, D. Ko, S. White, R. Flamini & T. Ketter: Felbamate monotherapy: implications for anti-epileptic drug development. Epilepsia 36, 1105-1110 1995 ; 298. Welty, T.E., M. Privitera & R. Shukla: Increased seizure frequency associated with felbamate withdrawal in adults. Arch Neurol 55, 641-645 1998 ; 299. Lammers, M.W., Y.A. Hekster, A. Keyser, H. van Lier, H. Meinardi & W.O. Renier: Neither dosage nor serum levels of anti-epileptic drugs are predictive for efficacy and adverse effects. Pharm World Sci 17, 201206 1995 ; 300. Koch, S., E. Jager-Roman, G. Losche, H. Nau, D. Rating & H. Helge: Anti-epileptic drug treatment in pregnancy: drug side effects in the neonate and neurological outcome. Acta Paediatr 85, 739-746 1996 ; 301. Koch, S., K. Titze, R.B. Zimmermann, M. Schrder, U. Lehmkuhl & H. Rauh: Long-term neuropsychological consequences of maternal epilepsy and anticonvulsant treatment during pregnancy for school-age children and adolescents. Epilepsia 40, 1237-1243 1999 ; 302. Samrn, E.B., C.M. van Duijn, S. Koch, V.K. Hiilesmaa, H. Klepel, A.H. Bardy, G.B. Mannagetta, A.W. Deichl, E. Gaily, M.L. Granstrm, H. Meinardi, D.E. Grobbee, A. Hofman, D. Janz & D. Lindhout: Maternal use of anti-epileptic drugs and the risk of major congenital malformations: a joint European prospective study of human teratogenesis associated with maternal epilepsy. Epilepsia 38, 981-990 1997 ; 303. Holmes, L.B., E.A. Harvey, K.S. Brown, A.M. Hayes & S. Khoshbin: Anticonvulsant teratogenesis: I. A study design for newborn infants. Teratology 49, 202207 1994 ; 304. Guerrini, R., A. Belmonte & P. Genton: Antiepileptic drug-induced worsening of seizures in children. Epilepsia 39 Suppl 3, S2-S10 1998 ; 305. Genton, P.: When anti-epileptic drugs aggravate epilepsy. Brain Dev 22, 75-80 2000 ; 306. Genton, P. & J. McMenamin: Aggravation of seizures by anti-epileptic drugs: what to do in clinical practice. Epilepsia 39 Suppl 3, S26-S29 1998 ; 148 and theo-dur.
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All of the following statements about cultures are true, EXCEPT: In cases of stromal corneal infiltrate of unknown origin, cultures are mandatory In such cases, the recommended diagnostic stain may be Giemsa Culture media including Sabouraud's dextrose agar, blood and chocolate agar are suggested In-office culture kits should not be used under any circumstances Which of the following statements about fungi is FALSE? Fungi are subdivided into yeasts unicellular ; and molds multicellular with filamentous hyphae ; Aspergillus species are the most common cause of fungal keratitis Candida infections are usually seen in cold climates Fusarium species are the most common fungi to cause keratomycosis in the United States The following statements about fungi are true, EXCEPT: They cause ocular morbidity via the infiltrative leukocytic inflammatory response Fungal toxins and enzymes cause secondary damage Trauma is the most commonly associated risk factor Treatment with corticosteroids is recommended from onset through to resolution of symptoms All of the following formed part of the treatment regimen in this case, EXCEPT: Ofloxacin Cuflox ; q.i.d. while awake In-office instillation of cyclopentolate hydrochloride Cyclogyl ; 1.0% Polysporin ophthalmic ointment at h.s. Naproxen sodium Naprosyn ; 500 mg at h.s. Regarding treatment of fungal keratitis, which of the following statements is TRUE? Amphotericin B 0.15% ; is an antifungal agent that has to be formulated for topical use Ofloxacin q. 1 hr. while awake is mandatory as adjunct therapy in the initial stages Fortified tobramycin Tobrex ; q. 30 min. should be alternated with fortified cefazolin q. 30 min. to treat fungal keratitis once a fungal culture is positive Polysporin ophthalmic ointment at h.s. is used to treat culture positive fungal keratitis.
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